Confirmed speakers
Prof. Christine Dijkstra, Vrjie University, Holland, Netherlands 
Innate immunity in Multiple Sclerosis
One of the world’s leading experts in macrophage biology who among other things first defined surface markers to enable macrophage phenotyping.
Prof. Uwe Hanisch, Göttingen University, Germany
Microglia - How versatile are their responses, how heterogeneous are they to start with?
One of the world’s leading experts in microglia biology who has recently characterized maturation states of microglia for the first time.
Prof. Hugh Perry, University of Southampton, UK
Microglia, cytokines and synapses in chronic neurodegeneration
A legend in the field of neurodegenerative diseases, including prion, dementias and inflammatory conditions.
Prof. Jennifer Pocock, University College London, London, UK
P53 family pathway activation in microglia; implications for neurodegeneration
Dr. Tobias Pukrop, Göttingen University, Göttingen, Germany
Carcinoma cells misuse the host tissue danger response to invade the brain
Prof. Marzia Malcangio, Kings College, London, UK
The role of microglia in positive and negative modulation of nociceptive fibre synaptic activity in the spinal cord
Prof. Bertil B Fredholm, Karolinska Institutet, Stockholm, Sweden
The role of adenosine A1 receptors for chemokine-mediated neuroprotection upon ischemic brain damage
Prof. Lars Farde, Karolinska Institutet, Stockholm, Sweden
Imaging macrophages and microglia in the CNS
Assoc Prof. Gunnar Schulte, Karolinska Institutet, Stockholm, Sweden
Proinflammatory transformation of microglia in response to WNT stimulation
Assoc Prof. Camilla Svensson, Karolinska Institutet, Stockholm, Sweden
The role of microglia in idiopathic pain
Assoc Prof. Robert Harris, Karolinska Institutet, Stockholm, Sweden
Myeloid cell therapy in neuroinflammatory diseases – future prospects for personalized medicine
A macrophage/microglia expert who address use of these cells in settings of Multiple Sclerosis, Stroke, Alzheimer’s disease, Pain, Brain tumors and Macular degeneration.
